0 Shop Products
Semax 10 mg research product by Vectrum Peptides
Neurobiological Signaling Research

Semax — 10 mg

10 mg / vial

Semax is a synthetic peptide based on part of the natural ACTH molecule. Researchers mainly use laboratory and animal models to study nerve-cell stress, communication and adaptation.

Sale 30% off €50,00€35,00
Research only Secure checkout Bac Water Needed
For in-vitro laboratory research only. Not for human or veterinary use.This information is provided for scientific and educational product context only. It does not describe or recommend human use, veterinary use, diagnosis, treatment, prevention, dosage, administration, or clinical application.

At a glance

What It Does in the Body

Focus and Attention

Semax is studied for brain signals involved in attention and focus.

Memory

Researchers study whether Semax affects signals used in learning and memory.

Mental Fatigue

It is connected to brain pathways involved in mental energy and fatigue.

Brain-Cell Signals

Semax is studied for signals that help brain cells adapt and communicate after stress or injury.

NECESSARY RESEARCH EQUIPMENT

Bacteriostatic Water

Available separately for laboratory use.

Full research details

Full research details for Semax

Read the supporting laboratory, animal and human research. This section does not provide instructions for human or veterinary use, diagnosis, treatment, prevention, dosage or administration.

What this compound is

Semax is a synthetic peptide based on a short fragment of ACTH, a natural hormone, but it is studied for different signalling questions rather than normal ACTH hormone activity. Research includes brain gene activity, BDNF-related signals and animal models of reduced blood flow. One human report also studied Semax during rehabilitation after ischaemic stroke.

MaterialSynthetic ACTH-fragment analogue
Main research areaNeural gene and stress-response research
Evidence availableCell, animal and limited human rehabilitation research
Catalogue categoryNeurobiological Signaling Research

How It Works

Normal target and function. Semax does not have one confirmed human receptor target. Preclinical work follows BDNF, a natural protein that helps nerve cells survive and adapt, and TrkB, the receptor that receives BDNF signals. Researchers also track broad gene responses after experimental brain-tissue stress.

Researchers use Semax models to ask how nerve cells change their gene activity after stress. One measured signal is BDNF, a protein involved in the maintenance and adaptation of nerve cells. Rat studies have examined TrkB, a receptor that responds to BDNF, and gene expression after experimental ischaemia. Separately, a 110-person post-stroke rehabilitation report measured blood BDNF, movement and everyday independence.

BDNF signals

Animal studies measure a nerve-cell support signal called brain-derived neurotrophic factor.

TrkB receptor

TrkB receives BDNF signals and is tracked in some rat brain experiments.

Gene activity

Researchers compare which genes become more or less active after an experimental challenge.

Stress models

Published sources include animal models of reduced blood flow to brain tissue.

Laboratory and Animal Research

Rat studies have reported changes in BDNF/TrkB-related signals and patterns of gene activity after experimental ischaemia or other controlled stress. These are molecular and animal observations, not evidence of better memory, mood, focus or recovery in people.

Measurements used in these studies

  • BDNF and TrkB measurements in selected rat brain regions
  • Changes in gene activity after controlled tissue stress
  • Differences between short-term molecular signals and lasting outcomes
  • Whether observations repeat across models and laboratories

Research Evidence

Human evidence

The sources used here are molecular or animal studies. They do not provide reliable controlled human results for memory, mood, attention, recovery or another body-level outcome.

Adverse effects

Reliable human adverse-effect information for this Semax research material is not established.

Evidence typeWhat was studiedHow much evidenceWhat this means
Laboratory researchGene-expression analysisPreclinicalStudies report changes in selected molecular signals under controlled conditions.
Animal researchRat brain and ischaemia modelsPreclinicalBDNF-related and gene-expression endpoints have been measured in rats.
Human researchPost-stroke rehabilitationEarly and limitedA 110-person report measured daily-function scores, movement and plasma BDNF. It did not test healthy focus, memory or energy.